Auxin Controls Megaspore Mother Cell (MMC) Initiation or Proliferation During Ovule Development

Mentor 1

Dazhong Zhao

Location

Union Wisconsin Room

Start Date

28-4-2017 1:30 PM

End Date

28-4-2017 4:00 PM

Description

In most flowering plants, a hypodermal cell at the tip of the nucellus differentiates to an archesporial cell then later forms megaspore mother cell (MMC) that accomplishes the transition from a somatic cell fate to a germline fate. However, how the MMC is formed and how the plant keeps only one MMC are still unclear. Our research focuses on elucidating the molecular mechanisms by which auxin controls megaspore mother cell (MMC) initiation or proliferation during ovule development via employing molecular genetics and cell biological techniques. Recently we found that application of auxin transport inhibitor and manipulation of auxin signaling that lead to disrupt the polarized auxin response and cause more MMCs. Thus, we hypothesize that the auxin maximum in the apical cells of nucellus provides a positional cue to restrict the somatic cells entering into the reproductive meiocyte. Moreover, activation of auxin signaling inside the nucellus will promote the MMC initiation or proliferation.



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Apr 28th, 1:30 PM Apr 28th, 4:00 PM

Auxin Controls Megaspore Mother Cell (MMC) Initiation or Proliferation During Ovule Development

Union Wisconsin Room

In most flowering plants, a hypodermal cell at the tip of the nucellus differentiates to an archesporial cell then later forms megaspore mother cell (MMC) that accomplishes the transition from a somatic cell fate to a germline fate. However, how the MMC is formed and how the plant keeps only one MMC are still unclear. Our research focuses on elucidating the molecular mechanisms by which auxin controls megaspore mother cell (MMC) initiation or proliferation during ovule development via employing molecular genetics and cell biological techniques. Recently we found that application of auxin transport inhibitor and manipulation of auxin signaling that lead to disrupt the polarized auxin response and cause more MMCs. Thus, we hypothesize that the auxin maximum in the apical cells of nucellus provides a positional cue to restrict the somatic cells entering into the reproductive meiocyte. Moreover, activation of auxin signaling inside the nucellus will promote the MMC initiation or proliferation.